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Desired fat threshold

Desired fat threshold

Deisred of Desired fat threshold impedance and BMI in predicting obesity-related medical conditions. What is visceral fat and why is it dangerous to your health? I lost 70 pounds on a commercial PSMF program before conceiving my first child.

Desired fat threshold -

Our growing understanding of the role of the liver and visceral fat has supported the development of the personal fat threshold hypothesis. This hypothesis states that everyone has a limit to the amount of fat they can store in the fat cells underneath the skin, which we call subcutaneous fat.

Collectively, this is also known as our adipose tissue. When this limit is reached, your body will start to store the fat within your vital organs including the pancreas and liver.

This transition from fat storage under the skin to the internal organs could give us a greater understanding of how type 2 diabetes develops and how to provide more individualised support for people living with this condition, regardless of their BMI.

This will be the focus of the rest of this article. Adipose tissue is mainly found beneath the skin in the form of subcutaneous fat. When you eat a meal containing fat, this fat will travel through the stomach to the gut.

We call these chylomicrons pronounced ky-low-my-crons. Chylomicrons are like boats as they transport fat the cargo around the body to the cells that need it. Depending on the requirements of each cell in the body, the fat will either be used for energy straight away or stored in the adipose tissue for later.

The smaller boat will then travel to the liver to be recycled. The liver will either store the leftover fat or send it out in other boats similar to chylomicrons known as low-density lipoproteins, or LDL.

LDLs perform a similar job as the chylomicrons, delivering fat around the body to either be used for energy or stored in the cells for later use. Imagine the gut to be the port, the chylomicrons the boat, the fat the passengers, and the liver the shipyard.

You can see this presented in the image below:. Glucose is then released into the bloodstream, which causes a rise in your blood glucose sugar levels. Your pancreas will respond to the rise in blood glucose by releasing the hormone to transport the glucose into the muscles and other cells of the body to be used for energy.

We call this hormone insulin. Insulin acts like a key; it opens the doors to the cells in your muscles to allow glucose to enter. The storage form of glucose is called glycogen.

The body can store around calories of glycogen in the muscles or around calories in the liver. Insulin also plays a vital role in this process. We mentioned earlier that fat travels around the body in boats called chylomicrons, and depending on what the cell needs, fat will either be stored in the adipose tissue or used for energy.

This means the body will use this glucose for energy when glucose is higher in the blood after we eat a carbohydrate-rich meal. The reason your body does this is because of the limited amount of storage you have for glucose.

In parts two and three of this series, we explain how the energy systems described above become dysfunctional and lead to you going beyond your personal fat threshold. Type 2 diabetes is a complex condition, and there are multiple theories about how it develops.

However, all theories agree that increased fat stored in the liver and pancreas plays a role. At this point, fat storage in the liver and pancreas will increase, and type 2 diabetes will develop or worsen.

Our body regulates and stores fat and carbohydrates through different systems. However, insulin plays a vital role in the regulation of both. Click here to read our next article in this series.

As seen on. All Blog Diabetes Exercise Lifestyle Mind Myth Busting Nutrition Recipes Sleeping. Diabetes What is your personal fat threshold? Written by Robbie Puddick Medically reviewed by Fiona Moncrieff 11 min read Last updated February Insulin and the personal fat threshold Insulin resistance and the personal fat threshold How to improve insulin resistance.

While not perfect, there are three ways we can measure this: 1 Waist circumference This estimates how much visceral fat you have stored in and around your internal organs. Again, the exception is in individuals with high muscle mass levels. Otherwise, keep reading as we dive deeper into the science behind the personal fat threshold.

The role of visceral fat The DiRECT study also reported significant reductions in pancreatic and liver fat in participants who achieved remission, regardless of the total amount of weight they lost.

We hypothesize that each individual has a personal fat threshold PFT which, if exceeded, makes likely the development of T2DM. Subsequent weight loss to take the individual below their level of susceptibility should allow return to normal glucose control.

Crucially, the hypothesized PFT is independent of BMI. So the personal fat threshold varies by individual, and does not tie directly to BMI measurements. This makes perfect sense, as the widely accepted Body Mass Index has several inherent flaws.

Highly muscular individuals for example, can often score as overweight - despite having an extremely low body fat percentage. As far as we can tell, your personal fat threshold is largely genetically determined. Based on the twin cycle hypothesis of the aetiology of type 2 diabetes and the theory of personal fat threshold , Roy Taylor and other researchers set out to imperically prove that a reduction in body fat would reduce the symptoms of type 2 diabetes , regardless of the subject's weight.

Using a 12 month study, they placed a test group of diabetics on an intense calorie-reduced diet. The results started to become apparent as early as the first week of the program.

Within 7 days, insulin resistance in the liver was essentially gone. Over the next 8 weeks, pancreas function also began to improve significantly for most of the test subjects. By the end of the 1 year study, the majority of recently diagnosed type 2 diabetics had completely recovered full insulin production, and were no longer displaying any diabetic symptoms.

Those who had lived with diabetes for 10 years or more prior to taking part in the study were less likely to see full recovery , but some did. Additionally, this study also proved that beta cells in the pancreas were not being directly killed off as previously believed - but merely going dormant.

This data was groundbreaking, but two issues remained. First, what were the long-term results for study subjects? And second, was a crash diet with massive weight loss absolutely necessary for success? Five years after the initial study, researchers collected follow-up data from the participants.

A number of the study subjects had regained some or most of the weight they lost during the study, and in most, type 2 diabetes returned with it.

Those who maintained their weight loss however, were twice as likely to have no further symptoms at all. Type 2 diabetes directly correllates with the personal fat threshold of the individual - which is largely dictated by genetic disposition.

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